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Similarly, GIP has an anti-inflammatory effect, as evidenced by the suppression of macrophage infiltration and the development of atherosclerosis in mice (Nogi et al

Spontaneous pemphigus vulgaris-like blisters associated with human IgG deposits were rarely found in mouse skin

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Diseases with an autoimmune component damage particular cells' mitochondria

6A), and ChIP analysis did not detect NRF2 binding in the CpG island (Fig

MicroRNA expression shows inflammatory dysregulation and tumor-like proliferative responses in joints of patients with post-infectious Lyme arthritis