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The glucose-dependent insulin secretion mechanism of GLP-1R and GIPR agonism means that insulin release is amplified only when blood glucose is elevated, reducing the risk of hypoglycaemia compared to insulin or sulfonylurea-based interventions

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Since the expression levels of oxidized glutathione trisulfide in the Si-DSS group were higher than those in the Con-DSS group, the GPx system was considered to be enhanced (Fig