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Compared to urinary PAHs metabolites, PAH-DNA adducts have a longer half-life in vivo, which can comprehensively respond to the exposure, uptake, metabolism, and DNA damage repair of PAHs in the organism 35

Rakesh Sahay has received speaker fees from Eli Lilly, Novo Nordisk and Sanofi

These intermediates induce mitochondrial oxidative stress by covalently binding to mitochondrial constituents including lipoylated dihydrolipoamide S-acetyltransferase (DLAT), leading to its aggregation and triggering regulated cell death via cuproptosis