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Why GLP-1 and GIP receptors are not the main drivers Mouse atrial preparation studies provided an important clarification
Subepithelial myofibroblasts expressing GLP-2R increase IGF-1 mRNA expression and secretion in response to GLP-2 stimulation, and genetic deletion of the intestinal epithelial IGF-1 receptor abolishes GLP-2-induced crypt cell proliferation in mice (69, 70)
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