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The mechanism underlying these gastrointestinal cytoprotection-associated findings has been attributed in published research to NO-related maintenance of mucosal microvascular function and preservation of endothelial integrity, rather than to simple acid suppression
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Thus, BPC 157 by naB2 regulation could represent a mechanism to guarantee a suited transient EGR-1 activity following injury [39]
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however, commercially viable product titres have not yet been reached, partly due to issues arising from the late steps of the biosynthetic pathway 6,7
Hematocrit, reticulocyte count, vitamin B12, folate and iron levels should be obtained prior to treatment
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