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By using a reducible disulfide linker, we built in a redox-sensitive release mechanism to facilitate intracellular release of the unmodified NMDA receptor antagonist, enabling the combined cellular actions of GLP-1 receptor agonism and NMDA receptor antagonism

1 HFpEF is a condition in which the heart pumps normally but is too stiff to fill properly, rendering the heart unable to support the bodys need for oxygen-rich blood

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