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6E-F), and these deficits were not significantly reversed by metformin in the Nrf2 KO mice (Fig
It was originally designed as a potential investigational treatment for neurodegenerative conditions, particularly Alzheimers disease
AAPS PharmSciTech 11(2):582587 Huang HC, Nguyen T, Pickett CB (2002) Phosphorylation of Nrf2 at Ser40 by protein kinase C regulates antioxidant response element mediated transcription
Regulating Glucose Metabolism MOTS-c has been shown to increase insulin-independent glucose uptake into skeletal muscle
A.GladyshevV
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