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Specific research has quantified these differences

This effect may be explained by a DPP-4 inhibitor-dependent reduction in intestinal lipoprotein production and consecutive lowering of circulating chylomicrons (36, 37)

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However, the magnitude of the effect of a deficiency in hypothalamic neuroendocrine signaling provides a clear direction for the development of therapeutic strategies

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In the subgroup analysis based on L-carnitine dose, when the treatment dose was more than 1 g/d, fasting blood glucose, triglycerides, and high-density lipoprotein-cholesterol were significantly improved