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Relationship of cytosolic glutathione to efflux and mitochondrial sequestration
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Since GSH is the final product of the homocysteine metabolism in the transsulfuration pathway by transferring sulfur from homocysteine to cysteine, a deficiency in transsulfuration pathway leads to excessive homocysteine production (hyperhomocysteinemia) and reduced GSH synthesis (159, 160)
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Its simultaneous action on GIP receptors further amplifies insulin secretion in response to food, while potentially improving the body's ability to utilize fat as fuel
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