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These changes were accompanied by a significant reduction in collagen deposition and overall fibrosis severity, suggesting that GLP-1 receptor agonism can modify established fibrogenic signalling cascades within the liver [38]

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Narrative review proposes pancreatic exocrine fibrosis and profibrotic signalling as key drivers of type 3c diabetes, with a potential role in type 2 and type 1 diabetes, and summarises unanswered questions & suggests potential novel therapeutic targets To view or add a comment, sign in / Metabolic dysfunction-associated steatotic liver disease (MASLD) is a major global health challenge due to its rising prevalence and strong association with metabolic syndrome, type 2 diabetes mellitus (T2D) and cardiovascular (CV) complications Effective management requires a comprehensive approach, emphasising aggressive treatment of obesity and T2D

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