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The importance of GPR119 in the fat-induced incretin secretion is demonstrated by the impaired incretin signaling displayed by transgenic animals deficient for this protein only in PG expressing intestinal cells

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While GLP-1R internalization was reduced after dual arrestin siRNA in MIN6B1-SNAP-GLP-1R cells (Supplementary Fig

TABLE 4 Pharmacologically underexploited peptides mediating bonemusclefat endocrine crosstalk: native peptide biology underdeveloped as therapeutic targets

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